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Nutritional counselling · Practice and clinic

Metabolic Health

What your client eats, how they process it — and what they can change.

Parameters
4 epigenetic + 17 genetic
Molecular level
Epigenetics and genetics
For tracking over time
the 4 epigenetic markers only
Genetic analysis
yes
Result format
Traffic light 1–9
Request a quoteThis report as PDFPDF in German
Sample report · Example dataReport for: Muster, ErikaSample collection: 08.04.2026Material: Dried blood spot cardEvaluations: 17 + 4 epigenetic
Overview
Metabolic Health

The other sample reports

Healthy AgingBiological Age ClockTelomere AnalysisStress MonitorHealthy Sport
The six panels compared

At a glance

Every value in this report

Each value with its classification. One click jumps to the full explanation below.

8 outside the range9 borderline9 within range

The principle

How to read this report.

This report combines two levels: your genetics — inherited and unchangeable — and your epigenetics, which can be influenced by lifestyle.

Level 1 · Genetics

Your genes are the blueprint of your metabolism: how you process carbohydrates, fats and proteins, how appetite and satiety work, how your body handles vitamins and salt. These predispositions do not change.

  • SNP — a well-researched variant in your DNA.
  • Three forms — wild type, heterozygous or homozygous; without any value judgement.
  • Your profile — several SNPs together produce your metabolic type and evaluations.

Level 2 · Epigenetics

Epigenetics does not show which genes you have, but how active they currently are. These four values can be changed — and are therefore the only ones in which the effect of an intervention can be demonstrated over time.

  • LINE-1 — global DNA regulation and stability.
  • Inflammation regulation — combined score from IL-6 and TNF-α.
  • miR-21 · miR-155 — microRNAs of metabolic activity.

Orientation

How do I read the scale?

All evaluations — genetic and epigenetic alike — use the same traffic light 1–9. Red on the left, green on the right: 1–3 red, 4–6 amber, 7–9 green. Higher is always more favourable.

13579
1–3 · Red
Outside the optimal range. This is where the greatest leverage lies.
4–6 · Amber
Moderate need for support. Monitor specifically.
7–9 · Green
In the favourable range. Maintain existing routines.

The miniature

The report in miniature.

This miniature shows in advance which result formats this report uses. It is calculated from the sample values on this page and is not a picture. Each mark stands for a format that reappears at full size further down — and whatever this panel does not use is missing here as well.

1159
  1. 1

    The traffic light 1–9

    Every evaluation sits as a mark on the same traffic light 1–9. Red on the left, green on the right, higher is more favourable. How the marks spread across the band shows directly whether this panel sits more towards the red, amber or green range.

Important notices

Sample report
All values, genotypes and personal details in this document are fictitious and serve solely to illustrate the report format. This is not a real report.
Interpretation
This report is not a diagnosis and does not replace medical advice. Genetic evaluations describe predispositions, not diseases; epigenetic markers describe a momentary state. Both are to be assessed in the individual clinical context by qualified professionals.
Status of the analyses
The analyses are laboratory services provided by our cooperation partner and are not CE-marked in vitro diagnostic devices. They serve consultation and monitoring and do not replace clinical diagnostics. The analyses are supplied to professional users only.
Legal framework
Genetic testing is subject to the German Genetic Diagnostics Act (GenDG); information and written consent are required before sample collection. The laboratory analysis is carried out by our cooperation partner. Polaris Diagnostics Europe GmbH is responsible for distribution and support. This document does not constitute any promise of cure within the meaning of the German Therapeutic Products Advertising Act (Heilmittelwerbegesetz).

Important

  • This report is not a diagnosis. Interpretation takes place in consultation with qualified professionals and always in the individual context.
  • Information and written consent are required before a genetic analysis (German Genetic Diagnostics Act, GenDG). Predictive genetic testing may only be conducted under the responsibility of appropriately qualified physicians; genetic counselling must be offered before and after the test (Sections 7 and 10 GenDG).
  • The genetic values never change. Only the four epigenetic values are suitable for monitoring over time.

How this becomes a consultation

A report is a programme, not a single value.

  1. 01

    Before the consultation

    The full report is available ahead of the appointment. Read it through and mark what stands out: the traffic-light ratings, their distribution across the evaluations, the starting points named. That way it is clear from the outset what to discuss.

  2. 02

    During the consultation

    Four values in this panel sit on the epigenetic level and can therefore be influenced. The genetic variants alongside them are determined once in a lifetime and remain unchanged; they describe the starting position. Only the epigenetic level responds to nutrition, exercise, sleep and strain — it alone is suitable for tracking progress.

  3. 03

    Afterwards

    A follow-up measurement is meaningful at the earliest after four to six months; before that, changes cannot be measured reliably. The interval is set by the supervising institution. For comparability, the laboratory, sample material and sampling should stay the same — and whatever happens between the measurements belongs in the same documentation as the values.

Your four modifiable values

4 values

Results at a glance

These four markers change through diet, exercise, sleep and stress — the reason for a follow-up measurement.

Your four modifiable values.
MarkerLevelYour valueOptimalInterpretation
LINE-1 · DNA stabilityMethylation2 / 97–9Outside range
Inflammation regulation (IL-6 & TNF-α)Methylation7 / 97–9Optimal
miR-21 · Inflammation & micronutrientsmicroRNA3 / 97–9Outside range
miR-155 · FTO regulation, adipose tissuemicroRNA8 / 97–9Optimal

Table scrolls sideways

Your metabolic type: balanced type

Your metabolism does not favour any one of the three macronutrients. The distribution and calculated energy requirement follow in the “Metabolic type” chapter.

Your exercise type: endurance type

Longer, steady exertion suits you better than short bursts of strength — a strength component nevertheless remains important.

What follows from this

  • Two red epigenetic values — LINE-1 (2/9) and miR-21 (3/9); the genetic MTHFR variant (2/9) depends on the same metabolic pathway. This is the central starting point of this report.
  • Two green values — inflammation regulation (7/9) and miR-155 (8/9) are in the optimal range and should be kept there.
  • A follow-up measurement is meaningful after 4 to 6 months at the earliest.

All genetic evaluations

17 values

Results at a glance

Seventeen evaluations on the traffic light 1–9. These values are inherited and do not change — they describe where your metabolism needs more support than average.

  • Skin hydrationAQPR9/9· Favourable
  • Difficulty maintaining weight8/9· Favourable
  • Salt sensitivity8/9· Favourable
  • Body weight regulation7/9· Favourable
  • Vitamin DTransport, GC7/9· Favourable
  • Oxidative cell protectionSOD27/9· Favourable
  • Food cravings6/9· Moderate
  • Skin elasticity & inflammationIL-66/9· Moderate
  • Difficulty losing weight5/9· Moderate
  • B vitaminsRequirement5/9· Moderate
  • Hunger/satiety regulation4/9· Moderate
  • Antioxidant capacityNADPH4/9· Moderate
  • Blood sugar and insulin metabolism3/9· Increased need
  • Blood lipids & fatty acid metabolism2/9· Increased need
  • Omega-3 supplyALA → EPA/DHA2/9· Increased need
  • FolateMTHFR2/9· Increased need
  • Weight loss through exerciseFTO2/9· Increased need

Sorted by value, not by topic. The detailed interpretation with genotypes and interventions follows in the chapters below.

Methylation

2 values

Epigenetic status

DNA methylation shows how active your genes currently are. Unlike the genes themselves, these values can be changed.

LINE-1DNA stability

LINE-1 elements make up around 17 % of the genome. Their methylation is regarded as a marker of global DNA regulation.

2 / 9Outside range
159

This marker is most effectively supported through the nutrients involved in folate metabolism — which gains additional importance given your MTHFR variant (genetic 2/9).

LINE-1 — Measure
MeasureSourceAmount / frequencyEffect
Folic acidgreen leafy vegetables, tomato, pulses, nuts, sproutsdailyIf needed, 2 months as methylfolate (5-MTHF)
Riboflavin (B2)germinated seeds, sprouts, cheesedailyCofactor of methylation
Cobalamin (B12)egg, cheese, fermented foodsdailyRegeneration of active folate
MethionineBrazil nuts, sesame, soya, peas, leafy vegetablesdailyDirect methyl group donor
Pyridoxine (B6)cabbage, green beans, lentils, bananasdailyCofactor of methylation
Fibrewholegrains, vegetables, pulsesat least 30 g/dayBioavailability of phytochemicals

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Inflammation regulation (IL-6 & TNF-α)

Combined score from the methylation of both gene regions — not the acute blood level. Individual values: IL-6 8/9 · TNF-α 6/9.

7 / 9Optimal
159

A plant-focused, low-inflammatory diet continues to support this area.

Inflammation regulation (IL-6 & TNF-α) — Measure
MeasureSourceAmount / frequencyEffect
Resveratrolgrape skins, raspberry seeds, blueberries, peanuts1 portion of berries/dayFound mainly in skins and seeds
Sulforaphanebroccoli, cauliflower, Brussels sprouts, cress3–4×/weekSteam only briefly or eat raw — otherwise the active substance is lost
Quercetinonions, garlic, capers, chivesdailyLet onions stand for 10 min after chopping
Curcuminturmeric with black pepper1 tsp dailyOil improves absorption; not during pregnancy or with gallstones

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microRNA

2 values

Epigenetic status

microRNAs fine-tune the activity of your genes and reflect current metabolic and inflammatory activity. Like methylation, they can be influenced through lifestyle.

miR-21Inflammation and micronutrients

Worsens with more pronounced inflammatory activity as well as with a poor zinc and folate supply.

3 / 9Outside range
159

With a red or amber value, a normal BMI should be aimed for; in addition, targeted zinc and folate intake is worthwhile. Antioxidant micronutrients such as vitamin D, vitamin E and lycopene as well as resveratrol support immune regulation.

miR-21 — Measure
MeasureSourceAmount / frequencyEffect
Zincpumpkin seeds, oats, lentils, cheese, beefdailyA poor zinc supply worsens this value
Folateleafy vegetables, pulses, tomato, sproutsdailyA poor folate supply worsens this value
Resveratrolgrape skins, blueberries, peanuts1 portion of berries/dayInhibits the NF-κB signalling pathway
Vitamin Dsalmon, butter, egg, avocado, mushroomsregularlyImportant for immune regulation
Vitamin Ewheatgerm oil, nuts, spinach, broccolidailyAntioxidant, protects cell membranes
Lycopenetomatoes, melon, carrots, papayadailyCarotenoid with an antioxidant effect

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miR-155FTO regulation and adipose tissue formation

Central regulator of immune and inflammatory responses; influences the formation of brown and beige adipose tissue.

8 / 9Optimal
159

Exercise, fasting and cold stimuli have a favourable effect here and influence the FTO effect positively — particularly relevant because your genetic FTO evaluation is 2/9.

miR-155 — Measure
MeasureSourceAmount / frequencyEffect
Regular exerciseendurance and strength combined150 min/weekHas a favourable effect on this value
Cold stimulicold showers, ice bathing, cool roomsregularly, brieflyActivates brown/beige adipose tissue
Fasting phasesextended overnight break from eatingafter consultationSupports the activation of brown adipose tissue
Quercetinonions, garlic, capers, chivesat least 200 mg/dayInfluences mitochondrial biogenesis

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Metabolic type: balanced type

3 values

Genetics · Macronutrients

Your metabolism does not particularly favour any one of the three macronutrients. The general recommendation of the DACH reference values therefore applies to you. More important in your case is the quality of the foods.

Metabolic type: balanced type.
ShareMacronutrientNote
55 %CarbohydratesWholegrains, pulses and vegetables as the basis
20 %Protein20–30 g per main meal
25 %FatFocus on unsaturated fats

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Individually calculated daily energy requirement: 1,900 kcal.

Exercise type: endurance type

2 values

Genetics · Exercise

Both ACE and ACTN3 point to a predisposition for endurance performance. For context: genetics explains only up to around 20 % of the difference between strength and endurance performance — training, diet and consistency have by far the greater influence.

Exercise type: endurance type.
GeneYour genotypePredisposition
ACEGG (homozygous)Rather endurance — variant associated with better oxygen utilisation and efficiency.
ACTN3TT (wild type)Rather endurance — XX type without α-actinin-3, more slow, endurance-oriented muscle fibres.

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Weight loss through exercise

1 value

Genetics · Exercise

Weight loss through exerciseFTO rs1121980

Genotype AT (heterozygous). How much exercise alone contributes to weight loss.

2 / 9Increased need
159

In your case, exercise alone leads to weight loss somewhat more slowly than with other genotypes. The effect on fitness and metabolic health remains unchanged — for weight reduction, the combination of exercise and dietary adjustment is particularly worthwhile.

Weight loss through exercise — Measure
MeasureSourceAmount / frequencyEffect
Endurance componentcycling, running, swimming2–3×/weekOxygen supply, basic endurance
Strength componentfull-body strength training2×/weekMaintains muscle mass and metabolic activity
Combine with dietsee metabolic type recommendationindividualWith FTO, the most effective additional lever

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Predisposition: body weight regulation and blood sugar metabolism

2 values

Genetics · Metabolism

Genetic predisposition for the regulation of body weight and for blood sugar metabolism — as a starting point for targeted nutrition, not as a prediction.

Body weight regulation

Summary of variants that are associated with the regulation of body weight.

7 / 9Genotypes: FTO rs9939609 TT · PPARG2 CC (wild type) · MC4R CT · TCF7L2 CT · SLC6A14 CA · TFAP2B GGFavourable
159

Your genetic background shows no unfavourable pattern here. Meal structure, fibre and regular exercise maintain this starting position.

Body weight regulation — Measure
MeasureSourceAmount / frequencyEffect
Fibrewholegrains, pulses, vegetablesat least 30 g/dayProlongs satiety, stabilises blood sugar
Protein per mealeggs, pulses, fish, meat20–30 g/mealSupports satiety signals
Exercise—150 min/weekAttenuates genetically driven effects

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Blood sugar and insulin metabolism

Genes that influence insulin secretion, insulin sensitivity and blood sugar metabolism.

3 / 9Genotypes: TCF7L2 CT · IL-6 CG · ADRB3 GG (wild type) · PPARG2 CCIncreased need
159

Your value lies outside the favourable range. Carbohydrate quality and regular exercise are the most effective levers in your case — and, together with the red miR-21 value, the second focus of this report.

Blood sugar and insulin metabolism — Measure
MeasureSourceAmount / frequencyEffect
Low glycaemic indexwholegrains, pulses, vegetablesevery main mealAvoids sharp blood sugar peaks
Movement after eatingshort walk10–15 minImproves insulin sensitivity

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Nutrition type

4 values

Genetics · Eating behaviour

Satiety, food cravings and the tendency to lose or maintain weight — your genetic levers around eating behaviour.

Food cravings

6 / 9MC4R CT · FTO rs9939609 TT · LEPR AA · SLC6A14 CA · PPARG2 CCModerate
159

A slightly increased tendency towards spontaneous eating impulses. Meal structure and protein intake are your most effective levers here.

Food cravings — Measure
MeasureSourceAmount / frequencyEffect
Protein with every mealeggs, pulses, fish, meat, tofu20–30 g/mealStrongest known satiety signal
Regular meals—3 fixed times/dayReduces blood sugar fluctuations

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Hunger and satiety regulation

4 / 9MC4R, LEPR and FTO rs1121980 (AT) togetherModerate
159

A somewhat delayed satiety signal — slow, mindful eating gives this signal the time it needs.

Hunger and satiety regulation — Measure
MeasureSourceAmount / frequencyEffect
Slow, mindful eating—at least 20 min/mealGives the delayed signal time
High-fibre dietvegetables, wholegrainsat least 30 g/dayProlongs the feeling of fullness

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Losing weight

5 / 9ADRB3 GG · PLIN CT
159

Increased need for support with sleep and leptin sensitivity. 7–9 hrs of sleep and combined training 2–3×/week.

Maintaining weight

8 / 9TCF7L2 CT
159

Favourable starting position against the yo-yo effect. Stable meal times and 150 min of exercise/week maintain it.

Vitamin uptake

3 values

Genetics · Micronutrients

How well your body utilises vitamin D and converts folate into its active form — including the resulting requirement for B vitamins.

FolateMTHFR

Genotype homozygous variant. Key enzyme in folate metabolism.

2 / 9Increased need
159

Your MTHFR variant indicates a markedly limited conversion of folate into its active form (5-MTHF). Your requirement for folate from food is higher than with other genotypes. This value also explains the red LINE-1 marker on the epigenetic level — both depend on the same metabolic pathway.

Folate — Measure
MeasureSourceAmount / frequencyEffect
Folate from foodgreen leafy vegetables, pulses, asparagus2–3 portions/dayMTHFR variants benefit from a consistently high intake
Active folate (5-MTHF)if required, as a food supplementafter consultation with a doctorBypasses the limited conversion directly

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Vitamin DTransport

GC (vitamin D-binding protein), genotype GT (heterozygous)

7 / 9Favourable
159

Vitamin D transport in your blood is only slightly limited. This does not mean a deficiency, but it does make a blood level check worthwhile during the months with little sunlight.

Vitamin D — Measure
MeasureSourceAmount / frequencyEffect
Vitamin Doily fish, eggs; sunlight15–20 min/day regularly, check levelsChecking is worthwhile above all in winter
Vitamin K2fermented foods, egg yolk, hard cheesedailySupports vitamin D in bone and vascular metabolism

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B vitaminsRequirement

Follows from the MTHFR variant via the methylation cycle.

5 / 9Moderate
159
B vitamins — Measure
MeasureSourceAmount / frequencyEffect
Vitamin B2eggs, dairy products, wholegrainsdailyDirect cofactor of the MTHFR enzyme
Vitamin B6potatoes, pulses, bananasdailyHomocysteine metabolism
Vitamin B12eggs, fish, dairy productsdailyRegeneration of active folate

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Omega 3 and blood lipids

1 value

Genetics · Lipid metabolism

How well you utilise plant-based omega-3 precursors and how your blood lipid levels are genetically predisposed.

Blood lipids and fatty acid metabolism

2 / 9FADS1 TT · APOA5 AG · LPL AA (wild type) · PPARG2 CCIncreased need
159

Your FADS1 variant indicates that the body converts plant-based omega-3 precursors (ALA) into the active forms EPA and DHA only to a limited extent. The separate evaluation “Omega-3 supply” looks exclusively at this conversion step and is likewise 2 / 9.

Blood lipids and fatty acid metabolism — Measure
MeasureSourceAmount / frequencyEffect
EPA and DHA directlyoily sea fish (salmon, mackerel, herring); alternatively algal oilfish 2×/week or algal oil dailyBypasses the limited FADS1 conversion
Plant-based omega-3 sourceslinseed oil, walnuts, chia seedsin addition, not as the sole sourceIn your case only partly converted
Limit omega-6sunflower, safflower and corn oil sparinglyin everyday lifeFavourable omega-6 to omega-3 ratio
Reduce sugar and white flourin favour of wholegrainsin everyday lifeRelates to triglyceride metabolism (APOA5)

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Salt sensitivity

1 value

Genetics · Blood pressure

Salt sensitivity

8 / 9ACE, genotype GG (homozygous)Favourable
159

Your ACE variant shows no increased salt sensitivity. As a general rule: anyone who eats a saltier diet should deliberately drink more.

Salt sensitivity — Measure
MeasureSourceAmount / frequencyEffect
Keep an eye on salt intakeprocessed foods, ready mealsmax. 6 g salt/dayReduces water retention
Drink enoughwater, unsweetened teaat least 1.5–2 l/daySupports sodium excretion
Potassium from vegetables and fruitbananas, potatoes, green vegetablesdailyCounteracts salt-related retention

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Nutricosmetics

4 values

Genetics · Skin and antioxidants

Skin elasticity, hydration and antioxidant protection — how diet supports your skin from within.

Skin hydration

9 / 9AQPR CC (wild type)
159

Your skin retains moisture well — a normal care routine is usually sufficient. Supportive: at least 1.5–2 l of water daily.

Oxidative cell protection

7 / 9SOD2 AG (heterozygous)
159

Solid protection against oxidative stress. Daily antioxidants and consistent UV protection maintain this starting position.

Skin elasticity and inflammation

IL-6 CG (heterozygous). Increased activity can accelerate collagen breakdown.

6 / 9Moderate
159

A moderate tendency towards inflammation in the skin — an anti-inflammatory diet has a visible effect on your skin.

Skin elasticity and inflammation — Measure
MeasureSourceAmount / frequencyEffect
Omega-3 fatty acidssalmon, linseed, walnutsfish 2×/weekSupports the skin barrier
Antioxidantsberries, colourful vegetables, green teadailyCounteracts oxidative stress

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Antioxidant capacity

NADPH AG (heterozygous). Provides the reducing power for cellular antioxidant systems.

4 / 9Moderate
159

A moderately reduced antioxidant capacity — antioxidants through the diet provide additional support here.

Antioxidant capacity — Measure
MeasureSourceAmount / frequencyEffect
Antioxidantsberries, colourful vegetables, green teadailySupports the cellular defence against free radicals
UV protectionsunscreen, headwearwhen exposed to the sunLimits UV exposure of the skin

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All genes analysed

21 values

Genetic basis

The complete list of the variants examined together with your genotype. These values are inherited and remain unchanged throughout life — repeating the genetic analysis serves no purpose.

All genes analysed.
GeneYour genotypeFunction
FTO rs9939609TT (homozygous)Risk allele A. Associated with BMI, body fat mass and waist circumference when the intake of saturated fatty acids is high.
FTO rs1121980AT (heterozygous)Risk allele A. Responds less well to weight loss through physical activity.
MTHFRhomozygous variantKey enzyme in folate and methylation metabolism; influences the homocysteine level.
FADS1TT (homozygous)Controls the conversion of plant-based omega-3/-6 precursors into the active, long-chain fatty acids.
APOA5AG (heterozygous)Regulates the breakdown of triglycerides in the blood and influences blood lipid levels.
LPLAA (wild type)Breaks down fats from the blood; influences HDL cholesterol and blood lipid levels.
PPARG2CC (wild type)Regulates fat cell formation, fat storage and insulin sensitivity; influences appetite and satiety.
PLINCT (heterozygous)Coats fat droplets in fat cells and regulates their build-up and breakdown.
ADRB3GG (wild type)Regulates fat burning in fat cells and the utilisation of stored fat.
MC4RCT (heterozygous)Centrally involved in controlling appetite, hunger and satiety signals in the brain.
LEPRAA (homozygous)Transmits the satiety signal of the hormone leptin.
SLC6A14CA (heterozygous)Transports amino acids; influences appetite, satiety and stress eating.
TCF7L2CT (heterozygous)Influences insulin secretion after carbohydrate-rich meals.
TFAP2BGG (wild type)Involved in the regulation of body weight and fat distribution.
IL-6CG (heterozygous)Central messenger substance of inflammation regulation; influences blood lipids, appetite and stress eating.
GCGT (heterozygous)Influences the transport of vitamin D in the blood.
ACEGG (homozygous)Part of the bradykinin-aldosterone system; influences endurance/strength predisposition and salt sensitivity.
ACTN3TT (wild type)Influences muscle fibre composition (fast vs. slow fibres).
AQPRCC (wild type)Influences water retention in the skin.
NADPHAG (heterozygous)Contributes to the antioxidant capacity of the cells.
SOD2AG (heterozygous)Important antioxidant enzyme, protects cells from oxidative stress.

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21 variants. Each of the evaluations in the chapter “All genetic evaluations” is derived from several of these genes together; a single genotype on its own does not allow any conclusion.

Summary

Your three focus areas.

From 21 genes and four epigenetic markers, three areas emerge in which effort and effect meet most clearly in your case.

01

Methylation metabolism — folate, B vitamins, zinc

Three values point in the same direction: LINE-1 2/9 (epigenetic), miR-21 3/9 (epigenetic) and folate/MTHFR 2/9 (genetic) — all three depend on the same metabolic pathway. This area is at the same time the only one whose effect can be demonstrated in a follow-up measurement. In concrete terms: green leafy vegetables and pulses daily, B2/B6/B12 via eggs, cheese and fermented foods, zinc via pumpkin seeds, oats and lentils. Active folate (5-MTHF) for two months after consultation with a doctor.

02

Fat quality — EPA and DHA directly

Blood lipids 2/9 and omega-3 supply 2/9. Your FADS1 variant makes plant-based omega-3 sources such as linseed oil and walnuts alone insufficient. Oily sea fish twice a week or algal oil daily bypasses this bottleneck directly; use omega-6-rich oils sparingly.

03

Blood sugar quality rather than calorie quantity

Blood sugar and insulin metabolism 3/9 alongside favourable body weight regulation (7/9). For you, the quality of the carbohydrates counts for more than the quantity: a low glycaemic index at every main meal, 20–30 g of protein per meal and a short walk after eating. Since exercise alone works more slowly in your case (FTO 2/9), this combination is the more effective route.

Follow-up and next step

  • A follow-up measurement of the four epigenetic markers is meaningful after 4 to 6 months at the earliest — before that, changes cannot be measured reliably.
  • The genetic evaluations remain unchanged and never need to be repeated.
  • A comparison of the measurement points over time is reported from the second measurement onwards.

Questions about your report?

Discuss the results with the facility that carried out the sample collection. That is where the clinical context needed for interpretation is available.

Contact

  • contact@polarisdx.net
  • +49 151 75011699
  • www.polarisdx.net

Distribution

  • Polaris Diagnostics Europe GmbH
  • Willy-Brandt-Straße 49
  • 20457 Hamburg

Sample report. All values, genotypes and personal details in this document are fictitious and serve solely to illustrate the report format. This is not a real report.

Interpretation. This report is not a diagnosis and does not replace medical advice. Genetic evaluations describe predispositions, not diseases; epigenetic markers describe a momentary state. Both are to be assessed in the individual clinical context by qualified professionals.

Status of the analyses. The analyses are laboratory services provided by our cooperation partner and are not CE-marked in vitro diagnostic devices. They serve consultation and monitoring and do not replace clinical diagnostics. The analyses are supplied to professional users only.

Legal framework. Genetic testing is subject to the German Genetic Diagnostics Act (GenDG); information and written consent are required before sample collection. The laboratory analysis is carried out by our cooperation partner. Polaris Diagnostics Europe GmbH is responsible for distribution and support. This document does not constitute any promise of cure within the meaning of the German Therapeutic Products Advertising Act (Heilmittelwerbegesetz).

© 2026 Polaris Diagnostics Europe GmbH · As at: August 2026

All six documents are sample reports: every value, genotype and personal detail is fictitious and serves solely to illustrate the report format. These are not real results.

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